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These dysfunctions collectively suggest that DJ-1 deficiency in microglia contributes to PD pathogenesis not only through enhanced neuroinflammation and oxidative stress but also potentially by compromising the intrinsic ferroptosis defense mechanism

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After the 12-week period, measurements of muscle GSH concentration increased by 340%, while TBARS decreased by 81%, and F2-isopropanes decreased by 80%, meaning decreased oxidative stress [48]